Translation of "Gliosis" in German

Gliosis, vascular proliferation and neuronal degeneration may be present in the brain.

Visual acuity increased gradually, but remained reduced due to epiretinal gliosis.

The histological investigations reveal intracytoplasmic and intranuclear inclusion bodies, necrosis of lymphatic tissues and interstitial pneumonia, while in the central nervous system may be observed demyelination, gliosis, neuronal degeneration, lymphocytes perivascular cuffing, leptomeningitis and inclusion bodies.

The authors conclude that subchronic exposures to a 900 MHz electromagnetic field signal for two months could adversely affect the rat brain (sign of a potential gliosis).

Postoperatively in two eyes an epiretinal gliosis developed, which made a second intervention necessary.

In the present study an accumulation of retinal kapillarektasia, intraretinal leakage and epiretinal gliosis is observed.

Four underwent surgery for epiretinal gliosis, one patient had proliferative diabetic retinopathy with retinal traction (PDR) without haemorrhage.

As much in brain, as in the associated nervous tissue to the intestine was intense infiltrated inflammatory mononuclear, an interstitial one and to perivascular with gliosis, neuronofagia and neuronal loss.

The authors conclude that chronic exposure could not induce cellular alterations such as proliferation, cell death (apoptosis) and reactive gliosis.

Reactive astrocytes were observed in connection with reactive gliosis in a series of pathological brain changes and neuropsychiatric diseases.

Reactive astrocytes were observed in connection with reactive gliosis in a number of pathological brain changes and neuropsychiatric disorders.

A major question is whether Müller glia subtypes and /or different pathomechanisms exit that lead to neuronal regeneration and/ or gliosis, respectively.

The surgical technique using the 25 gauge technique treating epiretinal gliosis is less traumatic than with other trocar guided methods and enables us to perform the procedure extremely with care.

Neuropathologically, all forms are characterized by neuronal loss, gliosis with astrocytic plaques and accumulation of tau-immunoreactive neurofibrillary tangles in specific brain areas.

Supposedly, these reach the retinal surface through retinal breaks and breaks in the internal limiting membrane and thus lead to epiretinal gliosis.

The data indicate that chronic exposure to GSM 900 MHz microwaves (SAR=6 W/kg) may induce persistent astroglia activation in the rat brain (sign of cellular change and a potential gliosis).

In 6 patients a vasculitis was demonstrated by fluoresceine angiography. Additional complications were epiretinal gliosis (n=3), macular oedema (n=4) and a choroidal neovascularisation membrane in both eyes (n=1).

Mean intravitreal levels were 3.08 µg/ml (range 1.54-3.45µg/ml) in patients with epiretinal gliosis and 33.3 µg/ml in the case with PDR.

4 of 34 eyes (=12%) developed a recurrent retinal detachment between 2 weeks und 27 months postoperatively. Also 4 of 34 eyes (=12%) showed a marked epiretinal gliosis. Vitreous hemorrhage, marked elevated intraocular pressure or visual acuity reducing subluxation or luxation of the IOL were not observed.

In the histopathological examination of the brain tissue, only slight differences were found between the sham exposed and exposed rats (mild degeneration in the glia, gliosis, vascular congestion).The DNA damage in the brain, as well as the mitotic index and the chromosome damage in the bone marrow cells were significantly increased in the exposed rats compared to the sham exposed rats.

Retinal kapillarektasia was found in 36 eyes intraretinal accumulation of fluorescein due to leakage in 26 eyes and epiretinal gliosis in 24 eyes.Conclusions: The importance of ocular findings like juvenile cataract, combined pigment epithelial and retinal hamartomas and optic nerve masses for the diagnosis of NF2 is certain.

Astrocytes perform diverse functions in the central nervous system (CNS), e.g. building up the blood brain barrier, regulating ion homeostasis and secreting molecules that affect other developmental processes in the CNS. Astrocyte functions play a crucial role when disturbed in pathological conditions, e.g. in reactive gliosis.